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Alzheimer's: neuronal receptor response may help explain memory loss

Parkinson's: gene therapy turns off mutation linked to disease

Parkinson's in Ashkenazi Jewish patients: LRRK2 gene mutation as major cause of disease

Stroke: reduced risk by eating more than five portions of fruit and vegetables a day

Alzheimer's: PAK defects appear sufficient to directly cause cognitive difficulties

Secondary strokes: severe stenosis of the arteries in the head is a major risk factor

Mild cognitive impairment exists in two different forms

Risk for Parkinson's disease associated with motor symptoms

Alzheimer's: region of DNA strongly linked to disease

Molecular pathway at synapses controls long-term memory

Immune cells help to maintain cognition and brain cell renewal

Reflex sympathetic dystrophy: nerve damage discovered

Reduced risk of dementia in older people linked to exercise

Vertebroplasty linked to increased risk for new fractures in adjacent vertebrae

Myelin breakdown linked to genetic risk factor for Alzheimer's disease

Brain: chemical signaling helps regulate sensory map formation

Laser technique sheds light on stroke

Endocannabinoids: Anandamide protects neurons from inflammation after brain damage

Severe intracranial stenosis, a major risk factor for the development of secondary stroke

Levetiracetam as adjunctive treatment in idiopathic generalised epilepsy patients with primary generalised tonic-clonic seizures

Neurons generated in the adult brain learn to respond to novel stimuli

Nervous system: the lack of Cbln1 might contribute to diseases

Parkinson's: restricting diet may reverse early-stage disease

Glatiramer may offer protection from axonal injury in patients with relapsing remitting multiple sclerosis

Inflammation linked to chronic pain

BDNF protein involved in neuropathic pain

Oxidative stress: EAAC1 protein appears to play a key role in protecting neurons

Cocaine might increase their risk of developing Parkinson's disease

Damage resulting from stroke: Neuregulin-1 protects brain cells

Breathing disturbances appear to originate from a deficiency in norepinephrine and serotonin in medulla in Rett syndrome

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